Endocannabinoid Regulation of Monoamines in Psychiatric and Neurological Disorders

Endocannabinoid Regulation of Monoamines in Psychiatric and Neurological Disorders

English | ISBN: 1461479398 | 2013 | 337 Pages | EPUB, PDF | 2 MB

The past decade has seen tremendous growth in the study of cannabinoid receptor signaling in brain. The impact and consequences of cannabinoid modulation of monoaminergic (dopamine, norepinephrine, and serotonin) circuits is becoming more clear. Scientists have shown significant interaction between these two systems in a variety of psychiatric and neurological disorders such as affective disorders, multiple sclerosis, and pain or pain disorders. The overarching goal of Endocannabinoid Regulation of Monoamines in Psychiatric and Neurological Disorders is to provide current information on advances in the field of endocannabinoid signaling and potential therapeutic applications with a particular emphasis on monoaminergic circuits.

Download:

http://longfiles.com/mgdvmgzue92e/Endocannabinoid_Regulation_of_Monoamines_in_Psychiatric_and_Neurological_Disorders.pdf.html

[Fast Download] Endocannabinoid Regulation of Monoamines in Psychiatric and Neurological Disorders


Related eBooks:
Food and Addiction: A Comprehensive Handbook
Multiple Sclerosis Immunology: A Foundation for Current and Future Treatments
Hepatitis A Virus in Food: Detection and Inactivation Methods
Molecular Pathogenesis of Colorectal Cancer
Sugar Toxic: The Dirty Fuel that Impairs Brain Function and Makes Us Fat
Power Sleep : The Revolutionary Program That Prepares Your Mind for Peak Performance
How to Change a Habit by Scott Young
When an Adult You Love Has ADHD : Professional Advice for Parents, Partners, and Siblings
Feed Your Face: Younger, Smoother Skin and a Beautiful Body in 28 Delicious Days
American Diabetes Association Complete Guide to Diabetes
Pathological Altruism
Naturally Pain Free: Prevent and Treat Chronic and Acute Pains-Naturally
Copyright Disclaimer:
This site does not store any files on its server. We only index and link to content provided by other sites. Please contact the content providers to delete copyright contents if any and email us, we'll remove relevant links or contents immediately.